Preview

Meditsinskiy sovet = Medical Council

Advanced search

Cervical endometriosis – solubility

https://doi.org/10.21518/2079-701X-2018-21-174-177

Abstract

Cervical endometriosis is a common disease in young women, which is characterized by the appearance of tissue on the vaginal part of the cervix, similar in structure to the endometrium and undergoing cyclic changes in accordance with the menstrual cycle, and is a form of external genital endometriosis. The article presents the pathogenetic mechanisms of this disease development in terms of possibility of exposure to them during pathogenetic therapy.

About the Authors

О. V. Kachalina
Federal State Budgetary Educational Institution of Higher Education «Privolzhsky Research Medical University» of the Ministry of Health of the Russian Federation
Russian Federation

Kachalina Olga Vladimirovna – Dr. habil., Associate Professor of the Department of Obstetrics and Gynecology of the Faculty of Additional Professional Education

603005, Nizhnij Novgorod, Minin and Pozharsky Square, 10/1



A. A. Korenkova
Federal State Budgetary Educational Institution of Higher Education «Privolzhsky Research Medical University» of the Ministry of Health of the Russian Federation
Russian Federation

Korenkova Anna Alexandrovna – postgraduate student of the Department of Obstetrics and Gynecology

603005, Nizhnij Novgorod, Minin and Pozharsky Square, 10/1



References

1. Damirov M.M. Modern tactics of management of patients with adenomyosis, a practical guide. M.: BINOM, 2015; 112 p. (In Russ).

2. Endometriosis: diagnosis, treatment and rehabilitation. Clinical guidelines. Moscow, 2013 (In Russ).

3. Ishchenko A.I. Pathogenesis, clinic, diagnosis and surgical treatment of common forms of genital endometriosis. Synopsis of a thesis … dr. habil. M. 1993 (In Russ).

4. Adamyan L.V., Kulakov V.I., Andreeva E.N. Endometriosis. M.: Medicine; 2006. 416 p. (In Russ).

5. Adamyan L.V., Spitsin V.A., Andreeva E.N. Genetic aspects of gynecologic diseases. M.: Medicine, 1998. 120 p. (In Russ).

6. Pashkov V.M., Lebedev V.A. Modern understanding of the etiology and pathogenesis of genital endometriosis. Gynecology, obstetrics and perinatology issues. 2007; 3: 52-61 (In Russ).

7. Gynecology: Textbook. Edited by Saveljeva G.M., Breusenko V.G. M.: GEOTAR-Media. 2012, 192 p. (In Russ).

8. Baskakov V.P., Tsvelev Yu.V., Kira E.F. Endometrial disease. SPb.: Publisher N-L, 2002. 452 p. (In Russ)

9. Damirov M.M. Genital endometriosis is a disease of active and business women. M.: Binom-Press; 2010. 192 p. (In Russ).

10. Sukhanova A.A. Genital endometriosis. Optimization of treatment of cervical pathology in women. Reproductive medicine. 2015; 1-2: 38-42. (In Russ).

11. Sampson J.A. Peritoneal endometriosis due to the menstrual dissemination of endometrial tissue into the peritoneal cavity. Am. J. Obstet. Gynecol. 1927; 14: 422-469.

12. Barbieri R.L. Endometriosis and the estrogen threshold theory. J. Repord. Med. 1998; 43(3): 268-279 [PMID: 19144942].

13. Apetov S.S., Apetov V.V. Etiopathogenetic aspects of endometriosis therapy. StatusPraesens. Gynecology, obstetrics, barren marriage. 2017; 3 (39): 91-98. (In Russ).

14. Amalinei C., Caruntu I.D., Giusca S.E., Balan R.A. Matrix metalloproteinases involvement in pathologic conditions. Rom. J. Morphol. Embriol. 2010; 51: 215-228. [PMID: 20495735].

15. Maybin J.A., Critchley H.O., Jabbour H.N. Inflammatory pathways in endometrialdisorders. Mol. Cell. Endocrinol. 2011; 335: 42-51.

16. Tariverdian N., Rucke M., Szekeres-Bartho J. e al. Neuroendocrine circuitry and endometriosis: progesterone derivative dampens corticotropin-releasing hormone-induced inflammation by peritoneal cells in vitro. J. Mol. Med. 2010; 88(3): 267-278.

17. Ishchenko A.I., Kudrina E.A. Endometriosis: diagnosis and treatment. M: GEOTAR-MED, 2002. 104 p. (In Russ).

18. Out-patient and ambulatory care guideline in obstetrics and gynecology, edited by V.E. Radzinskiy, M.: GEOTAR-Media, 2014. 944 p. (In Russ).

19. Dunselman G.A., Vermeulen N., Beker. C., Calhaz-Jorge C., D’Hooghe T., De Bie B. et al.; European Sosiety of Human Reprodution and Embryology. ESHRE guideline: management of women with endometriosis. Hum. Reprod. 2014; 29(3): 400-14.

20. Adamyan L.V., ed. Endometriosis: diagnosis, treatment and rehabilitation. Federal clinical guidelines for managing patients. Moscow: Russian Society of Obstetricians and Gynecologists; 2013. (In Russ).

21. Order of the Ministry of Health of Russia dated 01.11.2012 No. 572n (ed. Dated January 12, 2016) “On approval of the procedure for providing medical care in the profile “obstetrics and gynecology (except for the use of assisted reproductive technologies)” (Registered in the Ministry of Justice of Russia on 02.04.2013 No. 27960). (In Russ).

22. Oettel M., Carol W., Elger W., Kaufmann G., Moore C., Romer W., Klinger G., Schneider B., Schroder J., Sobek L., Walter F., Zimmermann H. A 19-norprogestin without 17(alpha)-ethinyl group II: Dienogest from a pharmacodynamic point of view, Drugs of Today. 1995, 31(7): 517–36.

23. Klipping C., Duijkers I., TA F., SF K, Schuett B. Pharmacodynamic study of four oral dosages of dienogest. Fertility and Sterility. 2010, 94(suppl 1, 4): S181, Abstract P-304.

24. Sasagawa S., Shimizu Y., Kami H. et al. Dienogest is a selective progesterone receptor agonist in transactivation analysis with potent oral endometrial activity due to its efficient pharmacokinetic profile. Steroids. 2008; 73(2): 222-231.

25. Miyashita M., Koga K., Takamura M. et al. Dienogest reduces proliferation, aromatase expression and angiogenesis, and increases apoptosis in human endometriosis. Gynecol. Endocrinol. 2014; 30(9): 644-648.

26. Tatsumi H., Kitawaki J., Tanaka K. et al. Lack of stimulatory effect of dienogest on the expression of intercellular adhesion molecule-1 and vascular cell adhesion molecule01 by endothelial cell as compared with other synthetic progestins. Maturitas. 2002; 42(4): 287-294.

27. Choi J., Jo M., Lee E., Lee D.Y., Choi D. Dienogest enhances autophagy induction in endometriotic cells by impairing activation of AKT, ERK1/2, and mTOR. Fertil. Steril. 2015; 104(3): 655–664.

28. Saito F., Tashiro H., Yamaguchi M., Honda R., Ohba T., Suzuki A., Katabuchi H. Development of a mouse model for testing therapeutic agents: The anticancer effect of dienogest on endometrial neoplasms. Gynecol. Endocrinol. 2016; 32(5): 403–407.


Review

For citations:


Kachalina ОV, Korenkova AA. Cervical endometriosis – solubility. Meditsinskiy sovet = Medical Council. 2018;(21):174-177. (In Russ.) https://doi.org/10.21518/2079-701X-2018-21-174-177

Views: 1089


Creative Commons License
This work is licensed under a Creative Commons Attribution 4.0 License.


ISSN 2079-701X (Print)
ISSN 2658-5790 (Online)